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Showing 1–9 of 9 results
Advanced filters: Author: "Andreas Diefenbach" Clear advanced filters

  • Mouse lymphoid tissue–inducer (LTi) cells require the transcription factor RORγt. Cupedo's group identifies RORγt+ human LTi cell equivalents as committed natural killer cell precursors, and teams led by Vivier and Diefenbach describe RORγt-expressing interleukin 22–producing natural killer cells in mouse gut.

    • Stephanie L Sanos
    • Viet L Bui
    • Andreas Diefenbach
    Research
    Nature Immunology
    Volume: 10, P: 83-91

  • The graded expression of the transcription factor T-bet in CCR6−RORγt+ innate lymphoid cells is found to be involved in the control of interferon-γ expression, a cytokine that is required to protect the epithelial barrier against Salmonella infections.

    • Christoph S. N. Klose
    • Elina A. Kiss
    • Andreas Diefenbach
    Research
    Nature
    Volume: 494, P: 261-265

  • Innate lymphoid cells (ILCs) have key roles in immune responses, lymphoid tissue development and tissue regeneration. Recently, several new ILC subsets were identified. Here, the authors propose the use of a uniform nomenclature to describe all ILC subsets to avoid confusion and further promote the development of this field.

    • Hergen Spits
    • David Artis
    • Eric Vivier
    Reviews
    Nature Reviews Immunology
    Volume: 13, P: 145-149

  • Genome-wide transcriptional profiling of tissue-resident innate lymphoid cells (ILCs) has provided important insight not only into their developmental relationships and phenotypic plasticity but also into previously unknown functions.

    • Andreas Diefenbach
    News & Views
    Nature Immunology
    Volume: 16, P: 222-224

  • It emerges that innate immune cells called group 3 innate lymphoid cells signal directly to intestinal stem cells to promote the replacement of damaged epithelial cells lining the gut. See Letter p.560

    • Konrad Gronke
    • Andreas Diefenbach
    News & Views
    Nature
    Volume: 528, P: 488-489

  • Sporadic inactivation of the interleukin-22 receptor in the intestinal epithelium of the mouse shows that IL-22 is required for effective activation of the DNA damage response following DNA damage.

    • Konrad Gronke
    • Pedro P. Hernández
    • Andreas Diefenbach
    Research
    Nature
    Volume: 566, P: 249-253