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Showing 1–3 of 3 results
Advanced filters: Author: "Jeffery D. Molkentin" Clear advanced filters

  • The use of mouse models has provided a wealth of data regarding the molecular mechanisms that control cardiac hypertrophy, ventricular remodelling and heart failure. Many of the signalling components involved are potential therapeutic targets for treating various forms of heart disease.

    • Joerg Heineke
    • Jeffery D. Molkentin
    Reviews
    Nature Reviews Molecular Cell Biology
    Volume: 7, P: 589-600

  • The heart undergoes physiological hypertrophy in response to developmental signals and increased workload. The structural and molecular characteristics of physiological cardiac hypertrophy are now being elucidated, as are the endocrine effectors and associated signalling pathways that regulate it.

    • Marjorie Maillet
    • Jop H. van Berlo
    • Jeffery D. Molkentin
    Reviews
    Nature Reviews Molecular Cell Biology
    Volume: 14, P: 38-48

  • Activated cardiac fibroblasts in adult hearts participate in the healing response after acute myocardial infarction and during chronic disease states. In this Review, Tallquist and Molkentin discuss insights gained from the use of genetically engineered mice that allow a systematic evaluation of fibroblast identity, origins, and response during cardiac disease and ventricular remodelling.

    • Michelle D. Tallquist
    • Jeffery D. Molkentin
    Reviews
    Nature Reviews Cardiology
    Volume: 14, P: 484-491