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Showing 1–5 of 5 results
Advanced filters: Author: "Prasad Devarajan" Clear advanced filters

  • New research supports the notion that pre-renal and intrinsic acute kidney injury are distinct molecular entities and hence different disease states despite similar increases in serum creatinine level. Pre-renal AKI induces protective molecular mechanisms whereas intrinsic AKI requires a 'second hit' that upregulates injury genes, and results in a persistent elevation of serum creatinine and kidney injury biomarkers.

    • Prasad Devarajan
    News & Views
    Nature Reviews Nephrology
    Volume: 13, P: 137-138

  • Acute kidney injury (AKI) is a common complication associated with increased patient mortality and progression to advanced chronic kidney disease. Mehrotra et al. recently investigated the incidence and long-term sequelae of AKI in a large cohort of kidney transplant recipients.

    • Edward J. Nehus
    • Prasad Devarajan
    News & Views
    Nature Reviews Nephrology
    Volume: 8, P: 198-199

  • Data are lacking on the impact of acute kidney injury (AKI) in children admitted to an intensive care unit. Schneider and colleagues have now performed a large-scale assessment of the use of the RIFLE criteria for characterizing AKI in children and have determined the mortality and morbidity associated with AKI in this patient group.

    • Stuart L. Goldstein
    • Prasad Devarajan
    News & Views
    Nature Reviews Nephrology
    Volume: 6, P: 393-394

  • Acute kidney injury (AKI) is diagnosed by the serum concentration of creatinine (SCr), an insensitive marker of kidney function. The authors of this Review discuss the properties of ideal biomarkers of tubular damage and how they can be combined with SCr-based AKI definitions to provide greater insights into the processes underlying acute tubular injury.

    • Beatriz Desanti De Oliveira
    • Katherine Xu
    • Prasad Devarajan
    Reviews
    Nature Reviews Nephrology
    Volume: 15, P: 599-612